BEGIN:VCALENDAR
VERSION:2.0
PRODID:icalendar-ruby
CALSCALE:GREGORIAN
METHOD:PUBLISH
BEGIN:VTIMEZONE
TZID:Europe/Vienna
BEGIN:DAYLIGHT
DTSTART:20180325T030000
TZOFFSETFROM:+0100
TZOFFSETTO:+0200
RRULE:FREQ=YEARLY;BYDAY=-1SU;BYMONTH=3
TZNAME:CEST
END:DAYLIGHT
BEGIN:STANDARD
DTSTART:20181028T020000
TZOFFSETFROM:+0200
TZOFFSETTO:+0100
RRULE:FREQ=YEARLY;BYDAY=-1SU;BYMONTH=10
TZNAME:CET
END:STANDARD
END:VTIMEZONE
BEGIN:VEVENT
DTSTAMP:20260404T110146Z
UID:5a3a343af0c9b480164818@ist.ac.at
DTSTART:20180406T150000
DTEND:20180406T160000
DESCRIPTION:Speaker: Nils Becker\nhosted by Gasper Tkacik\nAbstract: In mul
 ticellular organisms\, the cell cycle faces two unrelatedrequirements: wel
 l-defined cell size and adequate cell number. In the first part of the tal
 k\, I will argue that by measuring cell cycle durations in single-cell pro
 genies\, we can understand how the cell cycle regulatory machinery reconci
 les these two requirements. We found that cycle lengths are correlated bet
 ween distantly related pairs of cells within the same generation. This obs
 ervation is incompatible with simple models of inheritance of cycle-contro
 lling factors\; instead cycles are under the control of at least two antag
 onistic regulatory mechanisms. We identify the mechanisms as cycle progres
 sion and growth. The second part will be concerned with cell-cell communic
 ation in innate immunity. We study the type-I interferon system\, which ca
 n transmit multiple inflammatory messages via multiple cytokines but only 
 a single receptor.The unique biochemical architecture of this system can b
 e understood as a digital communication channel\, optimized for robust tra
 nsmission of a set of messages\, immune to concentration fluctuations.
LOCATION:Mondi Seminar Room 3\, Central Building\, ISTA
ORGANIZER:rgrah@ist.ac.at
SUMMARY:Nils Becker: FriSBi: Interferon codes in innate immunity / Hidden m
 emory in cell-cycle times
URL:https://talks-calendar.ista.ac.at/events/1204
END:VEVENT
END:VCALENDAR